Varied hypotheses have been offered to explain the difference in hypertension prevalence among Black and White Americans. Etiological hypotheses cover the gamut and implicate such diverse factors as slavery (Wilson & Grim, 1991), evolutionary adaptation to decreased availability of dietary sodium in the regions of origin (Wilson, 1986), pathogenesis of keloid fibroblasts (Dustan, 1995), and low birth weight (Lopes & Port, 1995). Although physiological-biological, nutritional, social, behavioral, and psychological factors have been implicated to explain the Black-White differential (Anderson, Myers, Pickering, & Jackson, 1989), no single factor appears to be sufficient to account for the prevalence rates. Rather, it is likely that the basis for the difference is multifactorially determined.