ABSTRACT
INTRODUCTION A (AOS) and childhood apraxia of speech (CAS) are acquired and congenital forms of a neuromotor disorder characterized by defi cits in the planning and execution of sequential speech movements in the absence of neuromuscular pathology (McNeil, Robin, & Schmidt, 2009). e childhood and adult forms of the disorder are not directly comparable because of diff erences in onset and etiology. However, they have been linked because of their speech defi cit similarities (Wambaugh, Duff y, McNeil, Robin, & Rogers, 2006; American Speech-Language-Hearing Association, 2007; Shuster & Wambaugh, 2008), and a presumed similarity in impaired neural mechanisms (Jacks & Robin, 2010).
