In keeping with the etiology theme, it also became apparent that the clinical aspects needed to be strictly separated from the animal aspects of zinc metabolism, a separation that has never previously been attempted. Although this division, like the separation of primary from secondary zinc depletion, may be somewhat arbitrary, it is the author‘s impression that current knowledge of the truly clinical aspects of zinc metabolism is too often confused with its effects in animals. The two will frequently be similar, but not always. In this book, therefore, animal studies are considered in part 2 (Biochemistry. Only a handful of references to animal studies has been included in part 1 (Clinical). The purpose of this separation is to clearly distinguish animal from human, experimental from clinical. Too many of the animal studies have involved severe and prolonged zinc deficiency or other exaggerated nutritional conditions that cannot be realistically applied in the clinical setting. Furthermore, animal studies are mainly or primary (dietary) zinc depletion, whereas in humans, secondary zinc depletion is more prevalent and has a more diverse etiology.

chapter 1|2 pages


part I|63 pages


chapter 1|5 pages


chapter 2|13 pages

Assessment of Zinc Nutriture

chapter 3|6 pages

Primary Zinc Depletion

chapter 4|17 pages

Secondary Zinc Depletion

chapter 5|3 pages

Acute Zinc Depletion

chapter 7|7 pages

Therapeutic Uses of Zinc

chapter 8|2 pages

Zinc Toxicity

part II|66 pages


chapter 9|10 pages

Zinc Homeostasis

chapter 10|4 pages


chapter 11|9 pages

Zinc Metabolism in Animals

chapter 13|11 pages

Fatty Acids, Lipids, and Membranes

chapter 14|1 pages


chapter 15|6 pages

Vitamins, Macroelements, and Trace Metals

chapter 16|3 pages


chapter 17|4 pages


chapter 18|3 pages

Physiology and Pharmacology

chapter |2 pages